Acute Coronary Syndrome

Coronary arteries are the portion of the heart muscles in which the blood flow reduces as a result of acute coronary syndromes that fail to function properly. For older patients, chest pain, nausea, vomiting, and diabetes mellitus are the most common symptoms associated with ACS.

ACS is most commonly associated with three clinical manifestations: names according to the Electrocardiogram (ECG) appearance. 1. Myocardial infarction with ST elevation (STEMI, 30 percent), 2. Non-ST myocardial infarction elevation (NSTEMI, 25%) 3. Diagnosis: Different methods of diagnosis • Electrocardiogram • Imaging and blood testing • Scores of prediction

Prevention: Acute coronary syndrome often reflects some degree of atherosclerosis damage to the coronaries. Primary atherosclerosis prevention controls the risk factors: healthy eating, exercise, hypertension and diabetes treatment, avoiding smoking and cholesterol levels; aspirin has been shown to reduce the risk of cardiovascular events in patients with significant risk factors. In myocardial infarction, secondary prevention is discussed.

Following the introduction of a smoking ban in all enclosed public places in Scotland in March 2006, hospital admissions for acute coronary syndrome were reduced by 17 percent. 67% of the decrease occurred in non-smokers.

Treatment: People with presumed ACS are typically treated with nitroglycerin, aspirin, clopidogrel or ticagrelor, and if morphine persists in the chest.


Reduction of Atherothrombosis for Continued Heath | Pathogenesis: Acute Coronary Syndrome | Molecular Mechanisms: Acute Coronary Syndrome | Immune System in Acute Coronary Syndrome | Myocardial Cell Death and Regeneration | Pharmacogenomics | Acute Ischemic Heart Disease | Echocardiography in Acute Coronary Syndrome | Nuclear Cardiology Techniques in Acute Coronary Syndrome | Plaque Passivation and Endothelial Therapy | The Modern Cardiac Unit | Antiplatelet Therapy | Anticoagulants